دسترسی رایگان به مقالات منتخب Gary Ruvkun برنده جایزه نوبل پزشکی (فیزیولوژی) ۲۰۲۴
مقالات مرتبط با جایزه
🔘Posttranscriptional regulation of the heterochronic gene lin-14 by lin-4 mediates temporal pattern formation in C. elegans, Cell (Dec 1993)*
🔘The C. elegans cell lineage and differentiation gene unc-86 encodes a protein with a homeodomain and extended similarity to transcription factors, Cell (Dec 1988)
🔘The unc-86 gene product couples cell lineage and cell identity in C. elegans, Cell (Nov 1990)
🔘Computational and Experimental Identification of C. elegans microRNAs, Molecular Cell (May 2003)
🔘The 20 years it took to recognize the importance of tiny RNAs, Cell (Jan 2004)
🔘Functional Proteomics Reveals the Biochemical Niche of C. elegans DCR-1 in Multiple Small-RNA-Mediated Pathways, Cell (Jan 2006)
🔘Detection of broadly expressed neuronal genes in C. elegans, Developmental Biology (Feb 2007)
🔘A Whole-Genome RNAi Screen for C. elegans miRNA Pathway Genes, Current Biology (Dec 2007)
🔘Tiny RNA: Where do we come from? What are we? Where are we going? Trends in Plant Science (July 2008)
🔘C. elegans Major Fats Are Stored in Vesicles Distinct from Lysosome-Related Organelles, Cell Metabolism (Nov 2009)
🔘Regulation of the C. elegans molt by pqn-47, Developmental Biology (Dec 2011)
🔘Inactivation of Conserved C. elegans Genes Engages Pathogen- and Xenobiotic-Associated Defenses, Cell (April 2012)
🔘The Caenorhabditis elegans RDE-10/RDE-11 Complex Regulates RNAi by Promoting Secondary siRNA Amplification, Current Biology (May 2012)
🔘Pseudomonas aeruginosa Disrupts Caenorhabditis elegans Iron Homeostasis, Causing a Hypoxic Response and Death, Cell Host & Microbe (April 2013)
🔘piRNAs and piRNA-Dependent siRNAs Protect Conserved and Essential C. elegans Genes from Misrouting into the RNAi Pathway, Developmental Cell (Aug 2015)
🔘Protein Sequence Editing of SKN-1A/Nrf1 by Peptide:N-Glycanase Controls Proteasome Gene Expression, Cell (April 2019)
🔘Mitochondrial Dysfunction in C. elegans Activates Mitochondrial Relocalization and Nuclear Hormone Receptor-Dependent Detoxification Genes, Cell Metabolism (May 2019)
🔘Hypoxia and intra-complex genetic suppressors rescue complex I mutants by a shared mechanism, Cell (Feb 2024)
🔘CMTR-1 RNA methyltransferase mutations activate widespread expression of a dopaminergic neuron-specific mitochondrial complex I gene, Current Biology (June 2024)
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